Lon1 protease – mycolab.ai

Autophagy and the Mitochondrial Lon1 Protease Are Necessary for Botrytis cinerea Heat Adaptation

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Scientists studied how a fungus that causes gray mold disease adapts to heat stress by examining two key cellular processes: autophagy (cellular cleanup) and a mitochondrial protease called Lon1. They found that both processes work together to help the fungus survive high temperatures by removing damaged cellular components and maintaining healthy mitochondria. When either process was disrupted, the fungus became much more vulnerable to heat and could not survive as well.

Autophagy and the Mitochondrial Lon1 Protease Are Necessary for Botrytis cinerea Heat Adaptation

mycolabadmin

Researchers studied how a common plant-damaging fungus called Botrytis cinerea survives high temperatures. They found that two cellular cleanup systems—autophagy (which recycles damaged components) and a mitochondrial protease called Lon1—work together to help the fungus survive heat stress. When either system was disabled, the fungus was much more sensitive to heat and showed increased cell death, suggesting these processes are essential for the fungus’s survival strategy.

Research Keyword: Lon1 protease

Autophagy and the Mitochondrial Lon1 Protease Are Necessary for Botrytis cinerea Heat Adaptation

Autophagy and the Mitochondrial Lon1 Protease Are Necessary for Botrytis cinerea Heat Adaptation