Research Keyword: Cell wall-degrading enzymes

Trichoderma and its role in biological control of plant fungal and nematode disease

Trichoderma is a beneficial fungus that can protect crops from diseases and pests while promoting healthier plant growth, without harmful chemical pesticides. It works through multiple strategies: competing with harmful fungi for nutrients, producing natural toxins that kill pathogens, directly parasitizing disease-causing organisms, and strengthening the plant’s own immune system. This eco-friendly approach reduces chemical pollution while improving crop quality and yields, making it an ideal solution for sustainable farming.

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Efficacy of Trichoderma longibrachiatum SC5 Fermentation Filtrate in Inhibiting the Sclerotinia sclerotiorum Growth and Development in Sunflower

Scientists studied a beneficial fungus called Trichoderma longibrachiatum that can fight a harmful mold (Sclerotinia sclerotiorum) which damages sunflower crops. They discovered that liquid containing products from this beneficial fungus can significantly stop the harmful mold from growing, prevent it from forming protective survival structures called sclerotia, and reduce its ability to infect plants. When tested on sunflower leaves, this fungal liquid was highly effective at both preventing disease before infection and stopping disease after infection had started.

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Effects of Thifluzamide Treatment on the Production of Cell Wall Degrading Enzymes in Rhizoctonia solani and Phenylpropane Metabolism in Pear Fruit

A new fungicide called thifluzamide was tested on pear fruits infected with a fungus that causes rot. The fungicide works in two ways: it stops the fungus from producing enzymes that break down plant cell walls, and it boosts the pear’s natural defense system by increasing protective compounds like flavonoids and phenolic acids. This dual action makes thifluzamide a promising treatment for preventing fruit rot during storage and transport.

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The Function of Chitinases CmCH1 and CmCH10 in the Interaction of Coniothyrium minitans and Sclerotinia sclerotiorum

Scientists studied two enzyme genes in a fungus that eats other harmful fungi. When they removed one gene at a time, the fungus still worked fine. But when they removed both genes together, the fungus grew slower and couldn’t attack its target fungus as effectively. This shows that these genes work together as a team to help the fungus do its job as a natural pest control agent.

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Physiological response of chestnuts (Castanea mollissima Blume) infected by pathogenic fungi and their correlation with fruit decay

This study examined how three types of fungal infections cause chestnuts to rot and decay. Researchers found that fungi produce enzymes that break down cell walls and trigger the fruit’s defense mechanisms, leading to tissue damage. When multiple fungi infect together, the damage is worse than individual infections. The findings suggest that reducing mechanical damage, using cold storage, modified atmosphere, and antimicrobial treatments can effectively prevent chestnut rot during storage.

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Optimization of cultural conditions for pectinase production by Diaporthe isolate Z1-1N and its pathogenicity on kiwifruit

Researchers studied how a fungus called Diaporthe causes soft rot in kiwifruit by producing special enzymes called pectinases that break down the fruit’s cell walls. They found the best conditions for growing these enzymes in the lab: a temperature of 28°C, neutral pH around 7.5, and 2-3 days of growth. When they extracted these pure enzymes and put them on fresh kiwifruit, the enzymes caused damage equivalent to about half the damage caused by the living fungus itself, proving these enzymes are important for disease development.

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Exogenous L-Arginine Enhances Pathogenicity of Alternaria alternata on Kiwifruit by Regulating Metabolisms of Nitric Oxide, Polyamines, Reactive Oxygen Species (ROS), and Cell Wall Modification

Researchers discovered that a specific nutrient called L-arginine paradoxically makes a fungus that causes black spot on kiwifruit more dangerous at low concentrations. The fungus uses this amino acid to trigger multiple survival mechanisms including producing protective molecules and enzymes that break down plant cell walls. However, at higher concentrations, L-arginine actually inhibits the fungus, suggesting it could be used as part of a disease control strategy.

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Study on Pathogenesis of Cytospora pyri in Korla Fragrant Pear Trees (Pyrus sinkiangensis)

Researchers identified Cytospora pyri as the fungus causing a serious disease in Korla fragrant pear trees in China. The fungus can only infect trees through wounds and spreads through the tree’s tissues over several weeks. Understanding how the fungus breaks down plant cell walls using enzymes could help develop better strategies to prevent the disease and protect pear orchards.

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Trichoderma: The Current Status of Its Application in Agriculture for the Biocontrol of Fungal Phytopathogens and Stimulation of Plant Growth

Trichoderma is a beneficial fungus that grows naturally in soil and can protect crops from harmful fungal diseases while promoting plant growth. It works through multiple mechanisms including directly attacking pathogenic fungi, competing for nutrients, and boosting the plant’s own defense systems. With over 77 commercial products already available, Trichoderma offers a promising sustainable alternative to chemical pesticides for protecting major world crops.

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Oxaloacetate anaplerosis differently contributes to pathogenicity in plant pathogenic fungi Fusarium graminearum and F. oxysporum

Two important crop-destroying fungi, Fusarium graminearum and F. oxysporum, rely differently on a metabolic enzyme called pyruvate carboxylase to cause disease. Researchers found that removing this enzyme completely eliminates the ability of F. oxysporum to infect tomato plants by blocking its capacity to penetrate roots and break down plant cell walls. However, the same enzyme deletion has minimal effect on F. graminearum’s ability to infect wheat, suggesting these fungi have evolved different metabolic strategies for attacking their hosts.

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