Cabbage wilt disease

The small GTPases FoRab5, FoRab7, and FoRab8 regulate vesicle transport to modulate vegetative development and pathogenicity in Fusarium oxysporum f. sp. conglutinans

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Researchers studied three important protein switches (Rab GTPases) in a fungus that causes cabbage wilt disease. By deleting these proteins one at a time, they found that each plays a critical role in fungal growth, spore production, and the ability to infect plants. The findings suggest that targeting these proteins could be a strategy to control the devastating cabbage wilt disease.

The small GTPases FoRab5, FoRab7, and FoRab8 regulate vesicle transport to modulate vegetative development and pathogenicity in Fusarium oxysporum f. sp. conglutinans

mycolabadmin

Scientists studied three proteins (Rab GTPases) in a fungal pathogen that causes cabbage wilt disease. These proteins act like traffic controllers, directing materials within fungal cells to support growth and disease spread. By removing these genes one at a time, researchers found that all three proteins are essential for the fungus to infect plants, produce spores, and survive stress conditions. This research could eventually help develop new ways to control this destructive crop disease.

Disease: Cabbage wilt disease

The small GTPases FoRab5, FoRab7, and FoRab8 regulate vesicle transport to modulate vegetative development and pathogenicity in Fusarium oxysporum f. sp. conglutinans

The small GTPases FoRab5, FoRab7, and FoRab8 regulate vesicle transport to modulate vegetative development and pathogenicity in Fusarium oxysporum f. sp. conglutinans