Anti-Therapeutic Action: cell wall degradation

Optimization of cultural conditions for pectinase production by Diaporthe isolate Z1-1N and its pathogenicity on kiwifruit

Researchers studied how a fungus called Diaporthe causes soft rot in kiwifruit by producing special enzymes called pectinases that break down the fruit’s cell walls. They found the best conditions for growing these enzymes in the lab: a temperature of 28°C, neutral pH around 7.5, and 2-3 days of growth. When they extracted these pure enzymes and put them on fresh kiwifruit, the enzymes caused damage equivalent to about half the damage caused by the living fungus itself, proving these enzymes are important for disease development.

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Integrated transcriptome and metabolome profiling reveals mechanisms underlying the infection of Cytospora mali in “Jin Hong” branches

This research examined how apple trees defend themselves against a serious fungal disease called Valsa canker caused by Cytospora mali. Scientists used advanced genetic and chemical analysis techniques to identify which genes and protective compounds are activated when apple branches are infected. They found that healthy apple trees fight the infection by strengthening their cell walls, producing special protective enzymes, and accumulating defense chemicals like α-linolenic acid and betaine. These discoveries could help develop better ways to prevent or manage this destructive disease in apple orchards.

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Integrated multi-omics identifies plant hormone signal transduction and phenylpropanoid biosynthesis as key pathways in kiwifruit (Actinidia chinensis var. deliciosa) resistance to Botryosphaeria Dothidea infection

Kiwifruit can be infected by a fungus called Botryosphaeria dothidea, which causes soft rot and makes the fruit inedible. Researchers used advanced techniques to study what happens inside the fruit when infected, finding that certain plant hormones and chemical pathways become active to fight the infection. They identified two key genes that appear to control how the fruit responds to the fungus, which could help develop better ways to prevent this costly disease.

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